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Noteworthy, EGCG only reduces cell proliferation and induces apoptosis in cancer cells but not in normal cells, demonstrating its invulnerability/safeness to be used as a potential alternative treatment ( Apart from producing promising effects, EGCG has several drawbacks, such as low stability, poor bioavailability, and highly metabolized under physiological conditions

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Importantly, there is no evidence that stressors such as SFN enable CRL Keap1 to release nonubiquitylated Nrf2 once the stressor has been eliminated from the cell [69, 264–266]