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As indicated above, S358 is the major phosphorylation site for hKOR and the S358N substitution totally abolished U50,488-induced receptor uncoupling from G proteins (Li et al., 2002)

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This study emphasizes on the preservation of intestinal barrier which plays a metabolic role in regulating inflammation in the intestines, which could eventually lead to management of adipose tissue dysfunction, chronic systemic inflammation, and glucose intolerance in T2D