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The iron-overload state commonly observed in AD brains exacerbates neuronal oxidative damage and, through the release of pro-inflammatory mediators and activation of immune cells, contributes to the structural disruption of the BBB

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The role of iron as a “micronutrient battleground” during viral infections, where ferroptosis may function as both a host defense and a pathway exploited by pathogens, offers new paradigms for antiviral therapies, suggesting we are only just scratching the surface of its intricate regulation