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Receptor VEGFR2 i odpowiedź śródbłonka W badaniach na komórkach śródbłonka i modelu niedokrwienia kończyny wykazano, że BPC-157 zwiększał ekspresję VEGFR2 oraz aktywację szlaku Akt-eNOS

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Edaravone showed potent antioxidant and radical scavenging activities in various pathologies, such as cerebral ischemia, stroke and amyotrophic lateral sclerosis [202,203,204], also improving oxidative stress-induced mitochondrial impairment in various diseases [205,206]